Team:Amsterdam/project/molecular design

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     $('#moldesign-content').append("This promoter derived from the wild-type version present in E. coli and has a modified AraI1 site, which cause this promoter to be less responsive to low concentrations of induction and exhibit a lower maximum response. The pBAD is very specifically activated by L-Arabinose. In the absence of arabinose the repressor protein AraC binds to AraI1 AraO2, blocking transcription. In the presence of arabinose, AraC binds to it and changes its conformation such that it interacts with the AraI1 and AraI2 operator sites, allowing transcription. For the characterization of our system, we choose the a weak version of the promoter, the pBAD-weak, taken from the parts registry (BBa_K206001).")
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Revision as of 19:19, 26 September 2012